![]() For this reason, several studies are oriented towards achieving more effective wound therapies, in order to reduce health costs and provide long-term relief and, ultimately, effective scar healing. ![]() The inconveniences caused by injuries, particularly for chronic wounds, are mainly related to treatment and management procedures limiting the wound repair, rather than tissue integrity restoration (so-called “restitutio ad integrum”). The repair process includes the interaction of cells, growth factors and cytokines involved in closing the lesion. Healing a skin wound shows an extraordinary mechanism of cellular function that is distinctive in nature. Severe skin damage can therefore be life-threatening. The skin is the largest organ in human body and plays a crucial role in different processes such as hydration, protection from chemicals and pathogens, vitamin D synthesis initialization, excretion and thermal regulation. Emerging skin regeneration techniques involving scaffolds activated with growth factors, bioactive molecules and genetically modified cells are exploited to overcome wound healing technology limitations and to implement personalized therapy design. Moreover, advanced treatment strategies for skin regeneration and experimental techniques for cellular engineering and skin tissue engineering are addressed. ![]() The main complications related to the healing process and the clinical management of chronic wounds are described in the review. The main goal of this comprehensive review is to focus on the progress in wound medication and how it has evolved over the years. Ongoing studies aim to obtain more effective wound therapies with the intention of reducing inpatient costs, providing long-term relief and effective scar healing. ![]() Physiological wound healing restores tissue integrity, but in many cases the process is limited to wound repair. ![]() Skin wound healing shows an extraordinary cellular function mechanism, unique in nature and involving the interaction of several cells, growth factors and cytokines. ![]()
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